So what is Emasculation?
The literal meaning of emasculation is the removal of the penis and the testicles, the external male sex organs. Removal of the testicles alone is termed castration. Emasculation was a form of punishment in Medieval Europe and sometimes formed part of the process of being hanged, drawn and quartered.
By extension, the word has also come to mean to render a male less of a man, or to make a male feel less of a man by humiliation. Needless to say, this metaphorical usage of the word is much more common than the application of its literal meaning.
In order to better understand this metaphorical usage, it’s necessary to take a look a masculinity.
Masculinity (also called boyhood, manliness, machismo, or manhood) is a set of attributes, behaviors and roles generally associated with boys and men. Masculinity is socially constructed, but made up of both socially-defined and biologically-created factors, distinct from the definition of the male biological sex.
Both males and females can exhibit masculine traits and behavior. Those exhibiting both masculine and feminine characteristics are considered androgynous, and feminist philosophers have argued that gender ambiguity may blur gender classification.
Masculine traits include courage, independence and assertiveness. These traits vary by location and context, and are influenced by social and cultural factors.
Masculine qualities, characteristics or roles are considered typical of, or appropriate for, a boy or man. They have degrees of comparison: “more masculine” and “most masculine”, and the opposite may be expressed by “unmanly” or “epicene“. Similar to masculinity is virility (from the Latin vir, “man”).
The concept of masculinity varies historically and culturally; although the dandy was seen as a 19th-century ideal of masculinity, he is considered effeminate by modern standards. Masculine norms, as described in Ronald F. Levant’s Masculinity Reconstructed, are “avoidance of femininity; restricted emotions; sex disconnected from intimacy; pursuit of achievement and status; self-reliance; strength and aggression, and homophobia.” These norms reinforce gender roles by associating attributes and characteristics with one gender.
In many cultures, displaying characteristics not typical of one’s gender may be a social problem. In sociology, this labeling is known as gender assumptions and is part of socialization to meet the mores of a society. Non-standard behavior may be considered indicative of homosexuality, despite the fact that gender expression, gender identity and sexual orientation are widely accepted as distinct concepts.
The relative importance of socialization and genetics in the development of masculinity is debated. Although social conditioning is believed to play a role, psychologists and psychoanalysts such as Sigmund Freud and Carl Jung believed that aspects of “feminine” and “masculine” identity are subconsciously present in all human males.
And now let’s take a look a femininity.
Femininity (also called feminity, girlishness, marianismo, womanliness or womanhood) is a set of attributes, behaviors, and roles generally associated with girls and women. Femininity is made up of both socially-defined and biologically-created factors. This makes it distinct from the definition of the biological female sex, as both males and females can exhibit feminine traits.
Modern conceptualizations of femininity also rely not just upon social constructions, but upon the individualized choices made by women:
Traits traditionally cited as feminine include gentleness, empathy, and sensitivity, though traits associated with femininity vary depending on location and context, and are influenced by a variety of social and cultural factors.
Tara Williams has suggested that modern notions of femininity in English speaking society began during the English medieval period at the time of the bubonic plague in the 1300s. Women in the Early Middle Ages were referred to simply within their traditional roles of maiden, wife, or widow.
After the Black Death in England wiped out approximately half the population, traditional roles of wife and mother changed, and opportunities opened up for women in society. Prudence Allen has traced how the concept of “woman” changed during this period. The words femininity and womanhood are first recorded in Chaucer around 1380.
In 1949, French intellectual Simone de Beauvoir wrote that “no biological, psychological or economic fate determines the figure that the human female presents in society” and “one is not born, but rather becomes, a woman,” just as implied above that one is not born, but rather becomes, a man.
This idea was picked up in 1959 by Canadian-American sociologist Erving Goffman and in 1990 by American philosopher Judith Butler, who theorized that gender is not fixed or inherent but is rather a socially defined set of practices and traits that have, over time, grown to become labelled as feminine or masculine. Goffman argued that women are socialized to present themselves as “precious, ornamental and fragile, uninstructed in and ill-suited for anything requiring muscular exertion” and to project “shyness, reserve and a display of frailty, fear and incompetence.”
Second-wave feminists, influenced by de Beauvoir, believed that although biological differences between females and males were innate, the concepts of femininity and masculinity had been culturally constructed, with traits such as passivity and tenderness assigned to women and aggression and intelligence assigned to men.
Girls, second-wave feminists said, were then socialized with toys, games, television and school into conforming to feminine values and behaviors. In her significant 1963 book The Feminine Mystique, American feminist Betty Friedan wrote that the key to women’s subjugation lay in the social construction of femininity as childlike, passive and dependent, and called for a “drastic reshaping of the cultural image of femininity.”
Behavior and personality
While the defining characteristics of femininity are not universally identical, some patterns exist: gentleness, empathy, sensitivity, caring, sweetness, compassion, tolerance, nurturance, deference, and succorance are traits that have traditionally been cited as feminine.
Femininity is sometimes linked with sexual objectification and sexual appeal. Sexual passiveness, or sexual receptivity, is sometimes considered feminine while sexual assertiveness and sexual desire is sometimes considered masculine.
An ongoing debate with regards to sex and psychology concerns the extent to which gender identity and gender-specific behavior is due to socialization versus inborn factors. According to Diane F. Halpern, both factors play a role, but the relative importance of each must still be investigated. The nature versus nurture question, for example, is extensively debated and is continually revitalized by new research findings. Some hold that feminine identity is partly a ‘given’ and partly a goal to be sought, just a as is the case with masculine identity.
In 1959, researchers such as John Money and Anke Erhardt proposed the prenatal hormone theory. Their research argues that sexual organs bath the embryo with hormones in the womb, resulting in the birth of an individual with a distinctively male or female brain; this was suggested by some to “predict future behavioral development in a masculine or feminine direction”. This theory, however, has been criticized on theoretical and empirical grounds and remains controversial. In 2005, scientific research investigating sex and psychology showed that gender expectations and stereotype threat affect behavior, and a person’s gender identity can develop as early as three years of age. Money also argued that gender identity is formed during a child’s first three years.
Mary Vetterling-Braggin argues that all characteristics associated with femininity arose from early human sexual encounters which were mainly male-forced and female-unwilling, because of male and female anatomical differences. Others, such as Carole Pateman, Ria Kloppenborg, and Wouter J. Hanegraaff, argue that the definition of femininity is the result of how females must behave in order to maintain a patriarchal social system.
In his 1998 book Masculinity and Femininity: the Taboo Dimension of National Cultures, Dutch psychologist and researcher Geert Hofstede wrote that only behaviors directly connected with procreation can, strictly speaking, be described as feminine or masculine, and yet every society worldwide recognizes many additional behaviors as more suitable to females than males, and vice versa. He describes these as relatively arbitrary choices mediated by cultural norms and traditions.
Hofstede describes as feminine behaviors such as “service”, “permissiveness”, and “benevolence”, and describes as feminine those countries stressing equality, solidarity, quality of , and the resolution of conflicts by compromise and negotiation.
Clothing and appearance
In Western cultures, the ideal of feminine appearance has traditionally included long, flowing hair, clear skin, a narrow waist, and little or no body hair or facial hair. In other cultures, however, expectations are different. For example, in many parts of the world, underarm hair is not considered unfeminine. Today, the color pink is strongly associated with femininity, whereas in the early 1900s pink was associated with boys and blue with girls.
These feminine ideals of beauty have been criticized by feminists and others as restrictive, unhealthy, and even racist. In particular, the prevalence of anorexia and other eating disorders in Western countries has frequently been blamed on the modern feminine ideal of thinness.
“In the second study, Reiner followed 12 genetically male children who were born with a similar defect, in that they lacked a penis at birth. All of these children underwent a sex reassignment surgery to appear female in infancy. Since then, 8 of the 12 have reassigned themselves back to a male identity. Of the the 4 who remain female, 3 of the sets of parents plan to tell their child about their genetic sex at birth “soon” and all 3 sets of parents expect that their children will switch back to a masculine identity after learning about their birth sex.
“Reiner comments on the results of the studies stating, “These studies suggest that male gender identity is directly related to normal male patterns of male hormone exposure in utero. These children demonstrate that normal male gender identity can develop not only in the absence of the penis, but even after the removal of testicles or castration at birth, and unequivocal rearing as female. Rather than the environment forming these children’s gender identity, their identity and gender role seem to have developed despite a total environment telling them they were female.” These and consequent other studies have caused the scientific community to reevaluate their belief that gender identity is constructed through “nurture”.
“Most scientists now believe that gender identity is something that is predetermined by a biological aspect and cannot be chosen for a child.
“This new outlook has called for the reevaluation of sex reassignment surgeries on infants and parents who are considering one for their child are urged with extreme caution to consider letting their child decide on their own at a later age. Reiner believes, “These studies indicate that with time and age, children may well know what their gender is, regardless of any and all information and child-rearing to the contrary. They seem to be quite capable of telling us who they are.”(2)
Male and Female Genetic Differences
There is an increasing amount of research information showing that the prenatal environment is critical to the presentation of gender identity. The dominating factor in this environment is the balance of hormones that the developing fetus is subjected to.
“Genetic information determines whether the primordial gonads, which are originally identical in males and females, develop as testes or ovaries. This is called sex determination. Once the gonads have developed as testes or ovaries, their hormonal products, particularly testicular hormones, determine physical development as male or female, a process called sexual differentiation. In human beings, the testes become active at about week 7 to 8 of gestation. Testicular hormones cause the external genitalia to develop in the male pattern (penis and scrotum), and in the absence of testicular hormones, the external genitalia develop in the female pattern (clitoris and labia). As outlined below, similar processes of sexual differentiation under the control of gonadal steroids occur in the mammalian brain during early development.”(4)
“Testosterone and estrogen are present in both males and females, with the principal active estrogen in both sexes being Estrodial (E2). The active portion of circulating E2 and of testosterone (T), is the free hormone, which is not bound to its specific plasma binding TBEG (testosterone-estrogen binding globulin or sex hormone binding globulin).
“The concentration of TBEG is sex hormone dependent, undergoing activation under estrogenic stimulation and suppression as a result of androgens. There is a dynamic antagonism between androgens and estrogens such that the biological effects of one may be inhibited by the presence of the other. Although the biochemical basis of this inhibition is unknown, its influence in disease states associated with devirilizing or feminizing changes is clear.”(14)
“Gonadal steroids have two general types of influences on brain and behavior, and they have been called organizational effects or influences and activational effects or influences. Organizational and activational effects are distinguished by their timing and their permanence. Organizational influences typically occur early in life, particularly prenatally and neonatally, and they are enduring. In contrast, activational influences occur later in life, typically in adulthood, and they are transient, waxing and waning as hormone levels rise or fall. An example is the activation of female sexual behavior by estrogen and progesterone near the time of ovulation, and the decline in sexual behavior at other phases of the cycle, when these hormones decline.”(4)
“Androgens, produced by the testes in males and the adrenal cortex in both sexes, appear to have a profound organizational effect on early neural development. Female rats exposed neonatally to androgens will mount other rats, which is a typical male mating behavior. Likewise, male rats deprived of androgen from an early age will exhibit lordosis, which is a typical female mating behavior of adopting the naturally occurring position for sexual receptivity. Males can be deprived of androgens by castration or prenatal exposure to alcohol or stress, which lower prenatal surges of testosterone in male fetuses. Castrated males are much less likely to behave sexually because of a lack of testosterone, a principle androgen. Administration of testosterone will increase the level of sexual activity exhibited by a castrated male. Castrated males also display an increase in lordosis.”(12)
“Several general principles of organizational influences are worth noting. One is that they typically occur during critical, or sensitive, periods of development, and these periods occur at times when testosterone is elevated in developing male animals. One implication of this general principle is that the hormone must be present at the appropriate time to have its effect. If it is present too early or too late, the impact will not be the same. In addition, although present only briefly, the impact of exposure to the hormone at the appropriate time can persist across the lifespan.
“Nevertheless, studies where hormones have been manipulated experimentally in a wide variety of mammalian species, ranging from rodents to rhesus monkeys, indicate that exposure to high levels of testosterone during critical periods of early development promotes male-typical neural and behavioral development, whereas exposure to lower levels of testosterone leads to female-typical neural and behavioral development. Given the large sex difference in human sexual orientation, the hypothesis that the early hormone environment influences this sexually differentiated outcome merits [increasing] investigation.(4)
Defeminization and Masculinization
“It has often been said by biologists that in sexual differentiation in mammals, the female is the “default” developmental pathway, in the sense that elimination of any of several gene actions necessary for formation of male genitalia leads to the development of external female genitalia (though development of functional ovaries requires effective action of several less understood sex-specific genes). Two processes: defeminization, and masculinization, are involved in producing male typical morphology and behavior. Disruption of either of these processes in males produces female-typical development. The opposite is not true, in that the disruption of normal sexual development in females does not lead to male-typical endpoints.
“Defeminization involves the suppression of the development of female typical morphology (development of the Müllerian ducts into the fallopian tubes, uterus and vagina) and behavioural predispositions. Masculinization involves the production of male typical morphology (development of the Wolffian ducts into male reproductive structures) and behavioral predispositions. Both defeminization and masculinization are required for a mammalian zygote to become a fully reproductively functional male.
“A brief version of the female default paradigm can be stated as follows:
- A set of specific genetic instructions must be present and a series of differentiating events mediated by hormones must occur in order for a mammalian zygote to become a fully reproductively functional male.
- The SRY, SOX9, and SF1 genes must be present and functional.
- Functional Leydig cells must form in the gonads.
- The Leydig cells must be able to produce testosterone.
- The target cells must have the hormone receptors to respond to the testosterone. The target cells of the external genitalia must have functional 5-alpha-reductase enzyme to convert some of the testosterone to more active dihydrotestosterone.
- There is some evidence that the brain must be exposed and respond to androgens either prenatally or early in life to produce characteristic mating behavior. This is well demonstrated in many animal species.
- To a large extent, each step builds on the previous. If anything goes wrong at any of the first four steps, the subsequent pathway of development results in female anatomy and behavior.
- No ovarian organizing gene homologous to SRY has been discovered. Both sexes are exposed to maternal estrogen prenatally. No hormones have yet been discovered that are necessary early in life to produce female sexual development. Estrogen seems not to be necessary until puberty for purposes of differentiation.
- The full development of male characteristics also includes personal experience throughout life, determining gender identity, gender roles and sexual orientation. However, there is much debate on the balance between nature and nurture in the determination.”(10)
Definition and assessment of sexual orientation in humans
“Sexual orientation typically is conceptualized as the direction of erotic interest – in males (androphilic), in females (gynephilic), or in both. In addition to these three categories, some individuals do not report erotic interest in either sex.
“It also is important to distinguish sexual orientation from other aspects of sexual identity, including core gender identity, and gender role behaviors. Core gender identity is the sense of self, usually as male or female, but occasionally as intersex or neither. Gender role behaviors are those behaviors that are culturally associated with one sex or the other. They also can be defined empirically as behaviors that show sex differences, meaning that they differ on the average for males and females in a given cultural group. These three categories of behavior might be expected to be consistent, but this is not always the case.
“Although many men understand themselves to be male, are interested in female sexual partners, and engage in male-typical behaviors in other areas, such as watching or playing football, this is not always so. The situation is similar for women, as well as for girls and boys. Behavior across these three categories, or across the many types of behavior included in the concept of gender role behavior, is not always consistent for a given individual. Nevertheless, there is some consistency, and, as will be discussed later in this chapter, the hormonal factors that influence sexual orientation also influence other aspects of sex-typed behavior, including childhood toy, playmate and activity preferences.”(4)
Do hormones have activational influences on sexual orientation in adulthood?
“Although both virilization of the external genitalia and neurobehavioral virilization depend on testosterone and its metabolites, there are many ways in which they can be decoupled. For instance, the time period when androgen influences development of the external genitalia is earlier in gestation than the time when most neurobehavioral effects are seen. Also, different co-factors are involved in masculinization of different neural regions, and similar differences in the co-factors whose action is required are likely to exist for the genitalia versus whatever neural systems are involved in hormonal influences on sexual orientation. Similarly, testosterone acts on the external genitalia largely following conversion to DHT, but may require conversion to estradiol before exerting some of its neurobehavioral effects.
Prenatal stress and sexual orientation
“Stress alters production of adrenal hormones, including testosterone and other androgens, raising the question of whether prenatal stress could influence sexual orientation. In rats, stressing pregnant animals increases female-typical sexual behavior in male offspring. These effects resemble those seen following reduction of androgen in developing male animals, for example by castration, and the effect of stress on development of male rats has been suggested to occur because it disrupts a prenatal surge of testosterone that occurs in typically-developing males.
“Possible reconciliations of the findings for humans versus rodents involve differences in the adrenal response to stress, and differences in the length of pregnancy. The adrenal response to stress appears to be less dramatic in humans than in rodents. In addition, human gestation and the period of prenatal sexual differentiation is far longer in humans than in rats, appearing to last for approximately 2 days in rats, but for months in humans. This additional time may allow feedback mechanisms in the human male fetus to adjust testicular androgen production to compensate for alterations in stress-related adrenal androgen secretion.”(4)
As with so many other physiological aspects of homeostasis, feedback mechanisms can ameliorate some of the common stress related disruptions to androgen secretion, which, for most gestating males, leads to normal sexual development.
Sexual orientation and childhood behavior
“Boys and girls differ, on average, in their toy, playmate and activity preferences. Boys tend to prefer toys like vehicles, such as cars, trains, trucks and airplanes, and weapons, whereas girls tend to prefer toys like dolls and tea sets. Sex differences in toy preferences appear in infancy, at least by the age of 12 months, and they grow larger as children develop into middle childhood. The mode of assessment (e.g., questionnaire versus observation) and the specific toys involved can influence the size of the sex differences, but behavioral observation of toy choices in childhood can show large sex differences for both masculine and feminine toys.
“A new study by researchers at the City University of London found that babies as young as 9 months old prefer to play with toys specific to their own gender.
“The experimenters, whose results were published in the journal Infant and Child Development this month, put 101 girls and boys between the ages of 9 and 32 months in a nursery with a variety of toys and observed which ones the children seemed to play with more.
“The toys used in the study were a doll, a pink teddy bear, a cooking pot, a car, a blue teddy, a digger and a ball. Toys were placed a few feet from the children so they would have to make some effort to get to them, and then researchers observed not only which ones they chose, but also how they played with them and for how long.
“Among the youngest, they found, the boys tended to prefer the ball and the girls wanted the pot.
“While modern parents like to think that everything is in our control when it comes to socializing children, the researchers found the opposite to be true. They write: “Our findings of sex differences in toy choice in the 9 to 17 months age group add some weight to the suggestion that such preferences appear prior to extensive socialization and do not depend on gender category knowledge but are reflections of our biological heritage.”
“So is our gender divide in toys entirely the product of nature? Obviously not.
“In fact, the researchers observed that babies and toddlers made almost no distinction between the blue and the pink teddy bears they were offered. Which suggests that certain things, like the color of a toy, only matter once kids are socialized to certain ideas — namely that pink is for girls and blue is for boys.(11)
“In regard to playmate preferences, 80-90% of children’s play partners are typically of their own sex. This sex difference also grows larger as children grow older; children at age 4.5 years spend about three times as much time with peers of their own than the other sex, whereas at age 6.5 years, they spend 10 times as much time with same sexed as with other sexed peers. In regard to activities, boys, like other male mammals, tend to engage in more rough and tumble play, involving overall body contact and playful aggression, than do girls. This sex difference is large for children playing with other children of the same sex.
“An individual’s sexual orientation typically becomes apparent after puberty. However, there is evidence that most men who are not heterosexual recall having been sex-atypical in their childhood behavior, particularly in their sex-typed toy, playmate and activity interests.
Hormones and sexual differentiation of childhood behavior
“There is substantial evidence that exposure to androgens prenatally influences children’s sex-typical toy, activity and playmate preferences. A consistent research finding, for example, is that girls who were exposed to high levels of testosterone prenatally, show increased male-typical toy preferences, playmate preferences and activity interests.
“These effects have been seen in studies conducted in a number of different countries in North America and Europe and using various methodologies, including interviews, questionnaires and direct observation of behavior. The results of these studies are consistent with the hypothesis that prenatal exposure to androgens influences the development of children’s sex-typical toy, activity and playmate preferences. Evidence from situations where women were prescribed hormones during pregnancy for medical reasons also support a role for androgens prenatally in the development of children’s sex typed interests. Children whose mothers took androgenic progestins during pregnancy show increased male-typical or decreased female-typical behavior, and those whose mothers took anti-androgenic progestins show the opposite outcome.
“Girls whose mothers took androgenic progestins while pregnant, typically are born with virilized genitalia, leading to suggestions that physical virilization could cause increased male-typical behavior. These suggestions have been addressed by showing that normal variability in androgen exposure prenatally is associated with normal variability in sex-typed toy, activity and playmate preferences postnatally.
Pathways to sexual orientation
“The factors leading to individual variability in most human psychological and behavioral characteristics are rarely singular. In most cases, several types of factors combine to produce a particular outcome. In addition, a number of different pathways can lead to a common outcome. Sexual orientation outcomes are likely to be similarly determined by multiple factors, and to have more than one multi-factored pathway leading to an outcome, such as androphilic orientation, gynephilic orientation or bisexual orientation.
“This article addressed the question of whether gonadal steroid exposure during prenatal development is one of the factors, in at least one of the pathways, that lead to variability in sexual orientation outcomes. Based on the compelling evidence that prenatal testosterone exposure influences children’s sex-typical play behavior, on the well-established links between childhood play interests and adult sexual orientation, and on the evidence showing altered sexual orientation in women exposed to high levels of androgens prenatally, the answer appears to be “yes”.
“It is worth discussing the reason for the link between sex-atypical toy, playmate and activity interests in childhood and sex-atypical sexual orientation in adulthood. A highly likely explanation is that the prenatal hormone environment influences both of these sex-related characteristics, childhood toy, playmate and activity interests and sexual orientation, directly, by acting during early development on the neural systems that regulate them.”(4)
Hormonal Production Pathways and Emasculation
Pregnenolone is the precursor to many other hormones.
17 hydroxy pregnenolone gets converted to DHEA.
DHEA can be converted to Androstenedione.
Androstendione can be converted to testosterone.
Testosterone is finally converted to dihydrotestosterone.
17 hydroxyprogesterone converts to 11 deoxycortisol.
11 deoxycortisol converts finally to cortisol.
Corticosterone converts to aldosterone which plays a role in fluid electrolyte balance.
Back to Androstendione which can also be converted to Estrone (E1).
Estrone can convert back and forth with itself and estradiol(E2).
E1 or E2 can covert to Estriol (E3), but this step is irreversible
Major Endocrine Hormone Pathways
LDL (Low-Density Lipoprotein)
17 Hydroxypregnenolone Progesterone
DHEA Corticosterone 17 Hydroxyprogesterone
↓ ↓ ↓
Androstendione Aldosterone 11 Dioxycortisol
Testosterone Estrone (E1) Cortisol
Dihydrotestosterone Estrodiol (E2)
Since cholesterol is the critical substance that the other steroid hormones are derived from, a low-cholesterol prenatal diet can lead to a reduction of the ideal hormone levels necessary for optimal sexual development in the fetus.
To make steroids we need an adequate amount of thyroid hormone (T3) and vitamin A around to convert the cholesterol to pregnenolone and then to DHEA. So if someone is hypothyroid and/or has low vitamin A levels they will be unable to produce optimal levels of pregnenolone and DHEA.
This means that all the steroids formed from cholesterol will also be lower if this continues chronically. That means the sex steroid hormones also fall, and so does Vitamin D levels.
The other affect of hypothyroidism is that it causes decreased steroid synthesis due to pregnenolone steal syndrome. Steroids secretion is how the brain maintains control over the twenty trillion cells in our bodies. If you can’t make the chemicals the brain needs to maintain this control, all hell can break loose from a control stand point.
Myelin Sheath Connection
Chemical Emasculation and Feminization
An increasing amount of research information is showing that the ingestion, absorption or inhalation of synthetic particular chemical substances drastically impacts the hormonal balance of male physiology and female physiology as well.
To the extent that these substances create an imbalance in the pregnant woman or cross the placental barrier into the fetus, they will also have negative effects on the hormonal balance and sexual development of the male or female. Once the child is born, the exposure to these substances will continue to impact the sexual development until maturity is reached.
Most of the research information has focused on the activational effects that occur from these chemical substances. As stated above, activational influences occur later in life, typically in adulthood, and they are transient, waxing and waning as hormone levels rise or fall.
• Industrial products and Plastics: • Bisphenol A (monomer for polycarbonate plastic and epoxy resin; antioxidant in plasticizers); • Phthalates (plasticizers); • DEHP (plasticizer for PVC); • Polybrominated biphenyl ethers (PBDEs) (flame retardants used in plastics, foams, building materials, electronics, furnishings, motor vehicles); • Polychlorinated biphenyls (PCBs);
• Food:• Erythrosine / FD&C Red No. 3; • Phenosulfothiazine (a red dye)• Butylated hydroxyanisole / BHA (food preservative); • Building supplies: • Pentachlorophenol (general biocide and wood preservative); • Polychlorinated biphenyls / PCBs (in electrical oils, lubricants, adhesives, paints);
• Insecticides: • Atrazine (weed killer); • DDT (insecticide, banned); • Dichlorodiphenyldichloroethylene (one of the breakdown products of DDT); • Dieldrin (insecticide); • Endosulfan (insecticide); • Heptachlor (insecticide); • Lindane / hexachlorocyclohexane (insecticide, used to treat lice and scabies); • Methoxychlor (insecticide); • Fenthion; • Nonylphenol and derivatives (industrial surfactants; emulsifiers for emulsion polymerization; laboratory detergents; pesticides);
• Other: • Propyl gallate; • Chlorine and chlorine by-products; • Ethinylestradiol (combined oral contraceptive pill); • Metalloestrogens (a class of inorganic xenoestrogens); • Alkylphenol (surfactant used in cleaning detergents; • 4-Methylbenzylidene camphor (4-MBC) (sunscreen lotions); • Parabens (methylparaben, ethylparaben, propylparaben and butylparaben commonly used as a preservative); •Benzophenone (sunscreen lotions);
“Their study lists the symptoms found in each of the numerous species tested, which includes testicular cancer, hermaphrodites, genital deformations, low sperm counts, and infertility.
“Numerous other studies over the past eight years confirm that similar dangers are posed to human health, and males are particularly at risk.
“Decreased sperm motility and concentration, as well as genital abnormalities in baby boys have been linked to these chemicals.
“The fact that endocrine disruptors like phthalates affect the reproductive system in nearly all mammals is becoming rather well known, although governments and public health- and safety organizations are slow to respond.
“The more a pregnant woman is exposed to high levels of phthalates, the greater the risk her son will have smaller genitals and incomplete testicular descent, leading to impaired reproductive development. The chemical also appears to make the overall genital tracts of boys slightly more feminine.
“It is believed that phthalates have these adverse effects because they reduce testosterone synthesis by interfering with an enzyme needed to produce the male hormone.
“This latest study adds to the growing body of evidence showing the harmful effects these chemicals have on males in particular.
“Women who had higher concentrations of two types of phthalates (DEHP, and DBP) also had boys who appeared more feminized in their personality, while playing.
“The presence of phthalates in the mothers was not associated with any differences in the girls’ play behavior. However, that is NOT an indication that phthalates are “safe” for women.
“They also affect women’s endocrine systems and reproductive health although the effects are different between the genders.”(8)
“Bovine growth hormones used in food production, soy foods, and bisphenol-A (BPA), just to name a few, add to the problem as they also mimic estrogen and disrupt your endocrine system.
“Other studies have linked the chemical to thyroid problems in both women and men, and researchers have also suggested a link between phthalates and illnesses like allergies, asthma, and contact dermatitis, all of which are on the rise in children.
Common Sources of Phthalates
“Phthalates are found in, among other things:
Processed food packaging
Vinyl flooring and wall coverings
Lubricant and adhesives
Beauty products like nail polish, hair spray, shampoo, deodorants, and fragrances
Other Gender-Bender Chemicals to Watch Out For
“Endocrine disrupting chemicals are everywhere these days and can be difficult to avoid unless you take decisive steps to limit or eliminate them from your immediate surroundings. This is particularly important if you are pregnant, or planning a pregnancy.
“Reading labels is a necessity these days. PVC plastics, which contain phthalates, will oftentimes carry the plastic recycling symbol # 3.
“Some of the other gender-bender agents to look out for include:
Bisphenol A (BPA) — a common ingredient in many plastics, including those in reusable water bottles and resins lining some food cans and dental sealants, can change the course of fetal development in a way that increases your risk of breast cancer.
Plastic items carrying recycling symbol # 7 is oftentimes indicative of a BPA-containing plastic.
Perfluorooctanoic acid (PFOA) — found in grease- and water-resistant coatings like Teflon and Gore-Tex, is a likely carcinogen.
Methoxychlor and Vinclozin— An insecticide and a fungicide respectively, have been found to cause changes to male mice born for as many as four subsequent generations after the initial exposure.
Nonylphenol ethoxylates (NPEs) — Known to be potent endocrine disrupters, these chemicals affect gene expression by turning on or off certain genes, and interfere with the way your glandular system works. They mimic the female hormone estrogen, and have been implicated as one reason behind some marine species switching from male to female.
Bovine growth hormones commonly added to commercial dairy have been implicated as a contributor to premature adolescence.
Soy products, which are loaded with hormone-like substances.
MSG — A food additive that’s been linked to reduced fertility.
Fluoride — This chemical in the U.S. water supply has been linked to lower fertility rates, hormone disruption and low sperm counts.”(8)
“Along with the overall health destroying effects of these chemicals is the feminization of males that has become so prevalent at the current time, although these effects have been building over the last 80 years, as more and more anti-testosterone disrupting chemicals have been introduced into the environment, the food supply and various products.
“These chemicals act to feminize men from the time they are conceived, since the average woman’s body contains close to 250 chemicals from cosmetics, household exposures, pesticides and from environmental pollution.
“All of these chemicals end up in women’s bodies since they’re stored in fat and women have more fat tissue than men in their breasts and their hips as well as their butt. When a woman becomes pregnant she begins to expose the male fetus to these chemicals.
“This happens through no fault of the woman, but through the oil, chemical and pharmaceutical companies, which produce and distribute these harmful chemicals throughout the world. Big Pharma has no incentive to reduce this exposure since they make huge profits on the ‘health care’ side of this situation by selling testosterone and Viagra to the millions of men who are emasculated.
“By producing and distributing BPA and atrazine, which are found in a variety of foods and products which are used by the public, and which find their way into our drinking water, the chemical companies can make millions of dollars of emasculating men and lowering their testosterone levels in order to make them impotent.
“As a result of this onslaught, sperm counts have decreased by 50%, while 85% of sperm cells are defective. Along with this, studies show that almost 30 million men are impotent in the US.
“The majority of this damage is done in the womb. Most of these chemicals destroy the cells within the testes that produce testosterone, called the Sertroli cells. So when males are exposed to these chemicals in the womb, they don’t end up with as many Sertroli cells as a normal fetus has, so their testosterone levels go down and they’re less able to produce sperm.
“So men today have less sperm produced than their grandfathers did and their testosterone levels are the lowest they’ve ever been in the history of mankind.
“This damage is done in the womb and there is no way to reverse this process of feminizing men, causing them to have undescended testicles, which is a major cause of infertility, often causing them to have hyospadias, with the urethra not at the usual location at the head of the penis and often causing the penis to be no longer than 2 inches, which can lead to a tendency to change their gender identity.
“This is a result of the fact that the sexual orientation and gender identity are formed in the womb and it’s not a choice that’s made to be bisexual, homosexual or heterosexual.
“What these chemicals do is block testosterone and since all human beings are first females and have the brain of females, when testosterone is blocked, the man becomes a male and looks like a male, with male genitals, but the result is that he has the brain of a female, because the testosterone never masculanized and caused the masculazation of the brain.
“So the net result is that the male animal behaves like a female, since they have the brain of a female and these chemicals can cause homosexuality.
“Since these effects take place in the womb, they can end up multi-generational through epigenetic processes, which results from the chemicals changing the DNA.”(9)
The effects of these chemicals on the brain also can lead to violent tendencies, while at the same time resulting in low motivation, lacking the inspiration to do anything, since testosterone levels are directly related to motivation.
These chemicals also cause adult men to look like pregnant women, with breasts and protruding bellies, since when testosterone is blocked and increase estrogen men develop belly fat which contributes to metabolic syndrome and in many cases type 2 diabetes.
This emasculation and feminization of males is all by design, since there is less need for men now that robots are taking over more and more manufacturing activities that were originally dominated by men. There’s less and less need for human labor, leading to the ability of an androgenous work force to perform the remaining industrial functions.
So the oligarchs have decided that the human population will be reduced with the emasculation and feminization of men being a critical part of the process, since it leads to sterility and a drastic decrease in the procreative instinct in humans.
The following video, The Disappearing Male, gives a good overview of the extent of the problem:
Deleted at Vimeo!!!
Cultural and Social Feminization
It’s clear from the above information that chemical feminization of men is the foundational cause of the increasing prevalence of feminized men in society.
Now it’s time to take a look at the cultural and social pressure on men to adopt a feminized mindset and societal presentation.
The Feminization of Public Education
This cultural programming has been occurring for at least as long a public education has existed.
Take at hazard one hundred children of several educated generations and one hundred uneducated children of the people and compare them in anything you please; in strength, in agility, in mind, in the ability to acquire knowledge, even in morality—and in all respects you are startled by the vast superiority on the side of the children of the uneducated.
— Count Leo Tolstoy, “Education and Children” (1862)
We want one class to have a liberal education. We want another class, a very much larger class of necessity, to forgo the privilege of a liberal education and fit themselves to perform specific difficult manual tasks.
domestic battleground for the scientific rationalization of social affairs through compulsory indoctrination. Great private corporate foundations led the way.
“Our form of compulsory schooling is an invention of the State of Massachusetts around 1850. It was resisted — sometimes with guns — by an estimated eighty percent of the Massachusetts population, the last outpost in Barnstable on Cape Cod not surrendering its children until the 1880s, when the area was seized by militia and children marched to school under guard.”
― John Taylor Gatto,
“We do not accept the traditional assumption that a woman has to choose between marriage and motherhood, on the one hand, and serious participation in industry or the professions on the other.”
“WE REJECT the current assumptions that a man must carry the sole burden of supporting himself, his wife, and family, and that a woman is automatically entitled to lifelong support by a man upon her marriage, or that marriage, home and family are primarily woman’s world and responsibility-hers, to dominate-his to support.”
“…the author of that book establishes beyond doubt that the woman who has always presented herself as a typical suburban housewife until she began work on her groundbreaking book was in fact nothing of the kind. In fact, under her maiden name, Betty Goldstein, she was a political activist and professional propagandist for the Communist left for over a decade before the publication of “The Feminist Mystique” launched the modern women’s movement.”
“Professor Horowitz documents that Friedan was from her college days, and until her mid-30s, a Stalinist Marxist, the political intimate of the leaders of America’s Cold War fifth column and for a time even the lover of a young Communist physicist working on atomic bomb projects in Berkeley’s radiation lab with J. Robert Oppenheimer.”